New PDF release: Molecular Biology of the Lung: Volume II: Asthma and Cancer

By Joanne T. Douglas, David T. Curiel (auth.), Prof. Robert A. Stockley (eds.)

ISBN-10: 3034887841

ISBN-13: 9783034887847

ISBN-10: 3034897731

ISBN-13: 9783034897730

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Extra info for Molecular Biology of the Lung: Volume II: Asthma and Cancer

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The activated form of NF-KB is a heterodimer, which usually consists of two subunits, p65 (RelA) and p50, although other forms such as ReI, RelB, v-ReI, p52, p105 (NF-KBl) and plOO (NF-KB2) may also occur. The p50 may be constitutively bound to DNA but requires p65 for transactivation activity. , where they play an important r~le in both development and primitive inflammatory responses. Targeted disruption of the genes coding for p65 or p50 in mice ("knockouts") results in severe immune deficiency which is lethal in the case of p65 [14, 15].

A specific IKB-a kinase complex (IKK) has now been identified and contains at least two interacting subunits [20]. Several signal transduction pathways are involved in the activation ofNF-KB and enzymes from the mitogen-activated protein (MAP) kinase pathways may interact at various points in the activation ofNF-KB [21]. A newly described kinase, NF-KB-inducing kinase (NIK), is a MAP3Krelated enzyme involved in the activation of IKK by tumour nectoris factora (TNF-a) and IL-l{3 [22].

The result showed only weak associations between HLA-DR allele frequencies and IgE responses to common allergens. A possible excess of HLA-DRI was found in subjects who were responsive to Fe! 006). Increased sharing of HLA-DRIDP haplotypes was seen in sibling pairs responding to both allergens. 1. Walley and W. O. C. M. Cookson Can II were not associated with any definite excess of HLA-DR alleles. No significant correlations were seen with the HLA-DP genotype and reactive to any of the allergens.

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Molecular Biology of the Lung: Volume II: Asthma and Cancer by Joanne T. Douglas, David T. Curiel (auth.), Prof. Robert A. Stockley (eds.)

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